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This analysis strengthens the increasing body of work identifying altered antioxidant responses as a potential contributor to cognitive impairment

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This temporal proximity means that metabolic perturbations are often detectable at lower drug concentrations and earlier time points than proteomic or transcriptomic changes making untargeted metabolomics a sensitive first-pass MoA screen

[,] REGULATION OF OXIDATIVE HOMEOSTASIS Redox status changes, such as elevated ROS or decreased endogenous antioxidant levels, are the pathological states seen in several neurological and psychiatric conditions